What is Mature Skin?

The aging process of the skin is a natural occurrence and progresses differently from person to person. The signs and appearance of aging and maturing skin will depend on your skin type and the type of external and internal environmental stresses your skin endures.
One of the major causes of aging is “oxidative stress.” This relates to damage to our DNA, proteins, and lipids (fats) caused by oxidants, which are highly reactive substances containing oxygen. These oxidants are produced normally but also result from inflammation, infection, and consumption of alcohol and environmental pollution.

The majority of age-dependent changes that occur in our skin happen in the dermis, which can lose from 20-80% of its thickness during the aging process. This is the result of changes in the fibroblasts, the cells responsible for collagen, elastin and glycosaminoglycan (GAG) biosynthesis. Not only is the collagen and elastin produced at a slower rate, which impacts the skin’s inability to repair itself, but the organization of the protein also changes, affecting the skin’s structure.

Most commonly changes to the skin appear when people reach their mid-twenties, their skin begins to change, and depending on its constitution, as well as external influences, signs of ageing will develop sooner or later. Typical changes include a loss of moisture and elasticity, causing tautness, and resulting attention and protection with enhanced skin care requirements.

Collagen play a crucial role and is what makes your complexion firm, plump, and youthful. This protein makes up 75 to 80 percent of your dermis.

Frequently Asked Questions

The effects of aging on the epidermis include the effect aging has on apoptosis and cell turnover rates. Expert studies indicate that the epidermal cell turnover rate slows from 30-50% between our thirties and eighties. Studies have also demonstrated that in young adults up to the age of 30, the Stratum Corneum transit time was as quick as 20 days, whereas in older adults it stretched to 30 plus days.
It has also become common knowledge that key elements that compound the process and cause additional havoc to our skin by producing free radicals, is the likes of excessive ultraviolet rays, smoking, pollution and stress. There are also other factors at play that relate to climate and man-made environments like air conditioned offices and homes. These free radicals have the power to help break down collagen fibers, causing the skin to thin and loose it youthful glow and texture.
Photoaging is characterized by fine and coarse wrinkles, skin dryness, skin laxity, pigmentation and vascular abnormalities. This includes sallow color and rough skin texture. Photoaging is caused by many years exposure to low levels of UV (ultraviolet ray). The result of long-term low-level UV exposure is the progressive thickening and wrinkling of the skin.
Changes in skin color are often associated with aging. Skin color is a composite of red, blue, yellow and brown coloration. This is the result of red oxygenated hemoglobin, yellow carotenoids and flavins and the brown melanin pigment of our skin. Hyperpigmentation spots are due to erratic melanocyte activity that is the result of cumulative Ultraviolet (UV) exposure. This is often associated with hypopigmentation (white spots), which also accompanies aging. The result is a mottled, older skin appearance made up of darker and depigmented areas. When we see an increase in yellow coloration in aged skin, it is the result of a decrease in brown melanin pigment along with a decline in red and blue-colored capillaries. In the case of cigarette smokers, the toxins cause a breakdown of elastin that also contributes to the yellow color of skin. This overall skin discoloration is often accompanied by an increase in broken veins. While hyperpigmentation is most often associated with skin aging, we also see hypopigmentation due to a reduction in the number of melanocytes; there is a decline of 6-8% per decade after age 30, which accounts for the lighter skin color.

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